Difference between revisions of "Abh"
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= This gene is a member of the following [[regulons]] = | = This gene is a member of the following [[regulons]] = | ||
{{SubtiWiki regulon|[[SigM regulon]]}}, | {{SubtiWiki regulon|[[SigM regulon]]}}, | ||
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{{SubtiWiki regulon|[[SigX regulon]]}} | {{SubtiWiki regulon|[[SigX regulon]]}} | ||
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=The [[Abh regulon]]= | =The [[Abh regulon]]= | ||
=The gene= | =The gene= |
Revision as of 08:39, 16 May 2011
- Description: transcriptional regulator of transition state genes
Gene name | abh |
Synonyms | ylxT, yzaA |
Essential | no |
Product | transcriptional regulator |
Function | regulation of gene expression during the transition from growth to stationary phase |
MW, pI | 10 kDa, 5.753 |
Gene length, protein length | 276 bp, 92 aa |
Immediate neighbours | mreBH, kinC |
Get the DNA and protein sequences (Barbe et al., 2009) | |
Genetic context This image was kindly provided by SubtiList
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Contents
Categories containing this gene/protein
transition state regulators, cell envelope stress proteins (controlled by SigM, W, X, Y)
This gene is a member of the following regulons
The Abh regulon
The gene
Basic information
- Locus tag: BSU14480
Phenotypes of a mutant
Database entries
- DBTBS entry: [1]
- SubtiList entry: [2]
Additional information
The protein
Basic information/ Evolution
- Catalyzed reaction/ biological activity:
- Protein family:
Genes controlled by Abh
Extended information on the protein
- Kinetic information:
- Domains:
- Modification:
- Cofactor(s):
- Effectors of protein activity:
- Localization:
Database entries
- Structure: 2FY9 (N-terminal DNA recognition domain)
- UniProt: P39758
- KEGG entry: [3]
- E.C. number:
Additional information
Expression and regulation
- Regulation:
- Regulatory mechanism:
- Additional information:
Biological materials
- Mutant:
- Expression vector:
- lacZ fusion:
- GFP fusion:
- two-hybrid system:
- Antibody:
Labs working on this gene/protein
Mark Strauch, Baltimore, USA homepage
Your additional remarks
References
The Abh regulon: PubMed
Other original publications
Other Publications: PubMed